Minerals are important for normal hematopoiesis and may play a role in acute hemolytic anemia induced by G6PD deficiency. 矿物质对正常血作用而言是重要的,而且其在G6PD缺乏造成之急性溶血性贫血可能扮演某些角色。
The clinical manifestations of these subjects were acute hemolytic anemia trigged by ingestion of fava bean and neonatal jaundice. 临床主要表现为新生儿黄疸、进食蚕豆后发生急性溶血性贫血等。
Objective To understand gene mutant types of glucose 6 phosphate dehydrogenase ( G 6 PD) deficiency among local Guangdong children and to evaluate the method using dried blood spots on filter paper in examination of patients suffered from acute hemolytic anemia caused by G 6 PD deficiency. 目的了解广东人葡萄糖6磷酸脱氢酶(G6PD)缺陷的基因突变型。
Objective: To explore the pathogenesis and clinical detection of acute hemolytic anemia ( HA) due to puerarin ( GGS). 目的:探讨葛根素注射剂(GGS)引起急性溶血性贫血(hemolyticanemia,HA)的不良反应的发生机制、临床特征和影响因素。
Relationship between Genetic Mutations of Glucose-6-phosphate Dehydrogenase Deficiency and Infantile Acute Hemolytic Anemia in Changsha City 长沙地区婴幼儿G6PD基因突变与急性溶血性贫血关系的研究
All these cases had acute hemolytic anemia, jaundice, hemoglobinuria and reduced G6PD activity to different extents. 所有患者都具有急性溶血性贫血,黄疸等临床表现及不同程度的血红蛋白尿、G6PD酶活性降低。
Conclusion: Postpartum HUS characterizes acute renal failure, microangiopathic hemolytic anemia and thrombocytopenia, usually occurring in a few days after an apparently normal pregnancy and delivery. 结论:正常妊娠及分娩后发生微血管病性溶血性贫血、急性肾功能衰竭、血小板减少为PHUS的临床特征。
Clinical detection of acute hemolytic anemia due to puerarin 葛根素注射剂致急性溶血性贫血临床分析
There were no significant differences between the clinical manifestations caused by the former two gene mutations, which both caused acute hemolytic anemia and jaundice. 前两种基因突变型所致的临床表现无明显差别,均引起新生儿急性溶血性贫血和黄疸。